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The self-medication hypothesis suggests that schizophrenia patients smoke, in part, to alleviate negative symptoms, dysphoric mood, and neurocognitive impairments by ameliorating a dysfunctional dopamine system.10 The addiction vulnerability hypothesis suggests that genetic and neurobiological factors associated with schizophrenia (ie, alterations in nicotinic acetylcholine receptors [nAChRs] and central dopamine systems) may predispose schizophrenia patients to nicotine addiction.11 Examining the effects of tobacco smoking on neurocognition in schizophrenia is crucial because it may help clarify the rationale for high consumption of tobacco products and inform treatment interventions

Example: In 2020, Utah passed HB 312 which specifically states post settlement funding does not meet the definition of a loan or credit

WYSACs model predicts that an additional $1.00 tax increase would decrease consumption by another 6%, or 0.31 packs per capita per month (Figure 5)

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