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Stimulation of GLP-1 receptor inhibits methylglyoxal-induced mitochondrial dysfunctions in H9c2 cardiomyoblasts: potential role of Epac/PI3K/Akt pathway

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Ten rats were randomly selected using a random number table to serve as the normal group, while the remaining 50 rats were designated as the Model group to establish the T2DM combined with MASLD model