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Excessive glial cell activation, which induces extensive proinflammatory cytokine release, contributes to the development of neurodegenerative diseases [46]

ZnT3, a membrane zinc transporter responsible for concentrating zinc into neuronal presynaptic vesicles and co-released with glutamate upon depolarization, is pivotal in maintaining neuron functions

Additionally, elevated homocysteine levels may indicate inefficient biosynthesis of essential proteins, caused by folate metabolism abnormalities

Whether they can be involved in the formation of kidney stones by inducing ferroptosis still needs to be further explored