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Chemotherapy-induced complement signaling modulates immunosuppression and metastatic relapse in breast cancer

In Vitro 1981, 17, 459466

When this balance collapses: Cells lose their ability to neutralize oxidative stress Mitochondrial efficiency declines Detox pathways become overwhelmed Cellular signaling shifts toward inflammation and degeneration This is where clinical outcomes begin to stall

Moreover, pathways mediating ferroptosis in breast cancer include T-cell inhibition via GCH1-BH4 [74] and ferroptosis triggered by DHODH-CoQH2 following GPX4 inactivation [75]