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[4] The result is a GHRH-receptor agonist that survives slightly longer in circulation than the unmodified hormone while still producing the same fundamental action: stimulation of the somatotroph cells of the anterior pituitary to release GH in a pulsatile, physiologic pattern, which in turn raises IGF-1

The net result is unprecedented weight loss that exceeds anything achievable through dual or single agonist mechanisms

In Graves disease, the immune system mistakenly creates antibodies that stimulate the thyroid gland to develop an excessive quantity of thyroid hormones, primarily thyroxine (T4) & triiodothyronine (T3)

New indications expand use cases Perhaps just as important as the wave of new GLP-1/GIP drugs for obesity is the expansion of already approved GLP-1 drugs into other therapeutic areas