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This results in a lower ability to promote receptor internalization compared with endogenous GLP-1, so it allows to have an increased GLP-1R expression on the cell surface, which translates in more robust insulinotropic properties, possibly explaining the enhanced insulin secretion (in both diabetic and not diabetic human islets), induced by TZP (48) ( Figure 1 )

Psychiatry 18 , 582594 (2013)

Pancreas cell physiology and pancreatitis cell biology: summary of a symposium held at the joint meeting of the EPC and the IAP, heidelberg 2002

Knowing these differences helps doctors give the best care, no matter where their patients are from