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Food consumption may impact the interlinked underlying factors associated with CKD, including inflammation, oxidative stress, mitochondrial dysfunction, and gut dysbiosis (Stenvinkel et al., 2020)

For those with hyperthyroidism, the goal is often to reduce inflammation and avoid excess iodine, which can fuel hormone overproduction

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Several mechanisms have been reported that explain the regulation of the concentration and activity levels of Keap1, such as transcriptional regulation (STAT6 and hypoxia-inducible factor) (59), miRNAs (miR-7 and miR-24-3p) (54), post-translational regulation, such as ubiquitination (60) and S-nitrosylation (61), as well as degradation of the Keap1 protein (p62-dependent autophagy) (62)