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The mutation in PP2B and PKC1 protein, involved in the neuronal plasticity in the cerebellum, leads to altered place cell activity or an unstable cognitive map in the hippocampus [181, 182]

Differential changes in PKLR in males (decreased) and females (increased) could have significant functional consequences in liver since glycolytic inhibition (such as by decreases in PKM2) has been suggested to promote the pentose phosphate pathway to generate higher levels of NADPH

What thoughts on that and where is that line

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