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Modulation or inhibition of HMGB1 release significantly reduces brain injury and improves neurological outcomes in various disease models, including ischemia, hemorrhage, trauma, epilepsy, and AD [255, 330]

nearly 80% live in low- and middle-income countries (Saxena and Li, 2017

Interestingly, they found aberrations in ROS clearance pathways, including elevated glutathione levels, decreases in mitochondrial superoxide dismutase levels, and glutathione peroxidase 4 mediated lipid oxidative damage

It uses ATP from mitochondria for this, and blocking P-gp with diltiazem brings back gemcitabines toxicity (Gu et al., 2022)