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Owing to complex formation, solvent-exposed Trp31 hTrx1 and Trp114 hTrxR1 fit into hydrophobic pockets in the interface region

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The kidneys high oxygen demand for blood filtration renders it exquisitely sensitive to redox imbalancean aberration common to both CKD and acute kidney injury (AKI) that, when coupled with iron dysregulation, unleashes ferroptosis: a non-apoptotic, iron-dependent form of regulated cell death driven by iron accumulation, lipid peroxidation, and antioxidant defense impairment (e.g., GPX4/SLC7A11 dysfunction), cascades to which the redox-sensitive kidney is uniquely predisposed

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