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Aldehyde dehydrogenase inhibition as a pathogenic mechanism in Parkinson disease

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Discussion In the current study, we revealed some unexpected interesting findings that chronic ethanol exposure reduced rather than enhanced T-cell hepatitis in Aldh2 -/- mice, which is in contrast to ethanol exacerbation of T-cell hepatitis in WT mice.13 Mechanistically, we found that acetaldehyde directly inhibited T-cell glucose metabolism and cytokine release, which contributed to the suppressed T-cell hepatitis in Aldh2 -/- mice after alcohol consumption

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