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During the last decade, it became evident that the long-term administration of MSG has deleterious effects on the kidneys by disturbing redox homeostasis, and inducing lipid peroxidation and histo-architecture lesions (Sharma, 2015)

1c, d) 42,58 , a rather unspecific GSH-dependent monothiol mechanism might be better suited for the reduction of a variety of physiological non-glutathione disulfide substrates to maintain metabolites and proteins in a reduced state (Fig