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Thus, in a nutshell, it is possible that the initial DNA damage that predisposes melanocytes to malignant transformation and cytogenetic alterations that later promote actual malignant transformation of the initially transformed melanocytes may be driven by dysregulation in melanin biosynthesis and by the increased cellular content of melanin [10]

We thank the Wellcome Trust and the Medical Research Council for funding, the European Union framework 5 and the Netherlands Organization for Scientific Research for support

GI: May cause bleeding, ulcers, or perforations

In the TME, immunosuppressive factors such as PD-L1, TGF-, and IL-10 suppress immune cell activity and promote immune evasion