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An explanation could be that the high GSH levels obtained by GSH1 overexpression prevent a further increase of the intracellular glutathione concentration, since it is known that Gsh1p is feedback inhibited by GSH[35]

The upregulation of a pantetheinase Vnn1 in adipose and muscle tissues of Cse / mice on the no-Cys diet suggests increased degradation of pantetheine into B5 and cysteamine 48 (Fig