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In the human renal glomerular endothelial cell line cultured under high-glucose and high-CHOL conditions, ABCA1 deficiency increases cellular CHOL deposition, leads to inflammation and apoptosis, disrupts the endothelial glycoconjugate barrier, and induces endoplasmic reticulum stress (ERS)

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Bakta: Rapid and standardized annotation of bacterial genomes via alignment-free sequence identification